One other test I overlooked in my translation from the the LEF blood test report to my blood work table was Lactate Dehydrogenase, which LEF / LabCorp labelled LDH. I'd only had this test done once before many years ago, and at that time it was labelled LD, so I didn't make the connection, and left LDH off my table. Here is an update, which I've also included in an edit to my full report above. It is actually pretty interesting:
- Lactate Dehydrogenase (LD/LDH) - 284 (RR 121-224). This one was well above the reference range. The one and only time I've had this test before was in 2002, when it was 187. For a 'normal' person elevated LDH might be cause for concern, as it may indicate the presence of HIV, cancer, meningitis or encephalitis. But I'm not a normal person (obviously) and elevated LDH is a sign of lactic acid metabolism, which can also result from endurance exercise - See below & PMID 7072633. I mentioned above that I'd done my usual 3h pre-breakfast exercise routine prior to getting the blood drawn for these tests - which could very well explain this elevated reading.
Regarding elevated LDH as a result of endurance exercise, study  tested the blood of well-trained runners (training up to 75 miles per week) both before and after a 13-mile run. Before the run, the average LDH of the runners was on average near the top of the reference range at 174 (RR 100-190), and 21% of them had LDH values above the upper end of the reference range. After the run, their mean LDH went up to 220, which is well above the top of the RR and 86% of the runners had values above the upper end of the RR, like I did.
I can hear all you exercise bashers chomping at the bit now - saying lactic acid is a harmful metabolic waste product. Dean's killing himself. Sorry, you're wrong. From this NY Times review of the evidence regarding lactic acid (my emphasis):
Everyone who has even thought about exercising has heard the warnings about lactic acid. It builds up in your muscles. It is what makes your muscles burn. Its buildup is what makes your muscles tire and give out...
But that, it turns out, is all wrong. Lactic acid is actually a fuel, not a caustic waste product. Muscles make it deliberately, producing it from glucose, and they burn it to obtain energy. The reason trained athletes can perform so hard and so long is because their intense training causes their muscles to adapt so they more readily and efficiently absorb lactic acid.
The notion that lactic acid was bad took hold more than a century ago, said George A. Brooks, a professor in the department of integrative biology at the University of California, Berkeley. It stuck because it seemed to make so much sense.
"It's one of the classic mistakes in the history of science," Dr. Brooks said....
Through trial and error, coaches learned that athletic performance improved when athletes worked on endurance, running longer and longer distances, for example.
That, it turns out, increased the mass of their muscle mitochondria, letting them burn more lactic acid and allowing the muscles to work harder and longer...
The understanding now is that muscle cells convert glucose or glycogen to lactic acid. The lactic acid is taken up and used as a fuel by mitochondria, the energy factories in muscle cells...
Just before a race, coaches often tell athletes to train very hard in brief spurts.
That extra stress increases the mitochondria mass even more, Dr. Brooks said, and is the reason for improved performance.
Mitochondria even have a special transporter protein to move the substance into them, Dr. Brooks found. Intense training makes a difference, he said, because it can make double the mitochondrial mass.
I strongly suspect an increase in mitochondria mass and an increase my mitochondria's ability to metabolize lactic acid for fuel as a result of my exercise and cold exposure routines are what enables me to run, bike and/or swim almost indefinitely (9+ hours per day) without getting tired or run out of energy, and also explains my elevated LDH.
[Update: Even more fascinating, subsequent to this post I discovered evidence not only that exercise and cold exposure increases lactate / lactic acid levels, but also that lactate results in the browning / beiging of subcutaneous white fat. See here for discussion.]
 Am J Clin Pathol. 1982 Mar;77(3):285-9.
Exercise-induced changes in common laboratory tests.
Priest JB, Oei TO, Moorehead WR.
Examination of 19 serum biochemical and hematologic parameters in a group of
white male runners, ranging in age from 23 to 47 years, just prior to and
immediately after a 13-mile "mini-marathon," demonstrated a significant increase,
by paired Student t-test, in mean values of: K+, BUN, creatinine, CK, LDH, AST
(SGOT), alkaline phosphatase, bilirubin, uric acid and leukocyte counts.
Prevailing environmental conditions were such as to produce no significant
hemoconcentration. Using this group's statistics and this hospital laboratory's
upper limits of normal, the percentage of values above two SDs are, for the
resting state: K+ 7%, BUN 7%, creatinine 0%, CK 21%, LDH 21%, AST 0%, alkaline
phosphatase 0%, bilirubin 7%, uric acid 7%, and leukocyte count 0%.
Post-exertional values above normal limits are: K+ 7%, BUN 21%, creatinine 21%,
CK 93%, LDH 86%, AST 0%, alkaline phosphatase 0%, bilirubin 14%, uric acid 36%,
and leukocyte 71%. Consequently, abnormally high values for K+, BUN, creatinine,
CK, LDH, bilirubin, uric acid, and leukocyte counts can often be expected in some
patients who exercise heavily. The degree of the abnormality will depend on the
level and length of exercise as well as the elapsed time between exercise and
There will never be peace in the world while there are animals in our bellies.